By Khalid Iqbal (Editor), Sangram S. Sisodia (Editor), Bengt Winblad (Editor)
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Extra resources for Alzheimer's Disease: Advances in Etiology, Pathogenesis and Therapeutics
This instrument assesses multiple cognitive abilities, including orientation as to time and place, immediate and delayed word recall, naming, verbal repetition, reading, writing, and spatial ability. , 2000). This result supports the view that the earliest cognitive deﬁcits in AD are seen within the domain of episodic memory. 2. , 2001) reported the results from a comprehensive neuropsychological battery administered to a sample of the entire Kungsholmen Project’s cohort. This sample was examined three times during a follow-up period of six years.
Unfortunately this does not appear to be true. , 1997)] indicates that the exponential increase of the prevalence of dementia continues well past 85, apparently up to age 95. As the prevalence of dementia may exceed 50% after age 95, there must then be a plateau, since there certainly are centenarians with intact cognitive functions. Determining what happens at very advanced ages will be a major objective for future epidemiological studies. DIAGNOSING VERY MILD CASES OF DEMENTIA IN EPIDEMIOLOGICAL STUDIES Most of the prevalence studies cited have used either Mini-Mental State Examination (MMSE) scores of 24 or lower or equivalent scores on related tests as a screening tool.
VASCULAR RISK FACTORS Many clinicians have been struck by the seeming good health of individuals who develop AD. , 1990). Sparks et al. (1990) reported a high correlation between coronary stenosis and cerebral plaques in a medical examiner autopsy series. ApoE-e4 predisposes to myocardial infarcts in young adult males and AD later in life. , 1997) may alter both brain amyloid and the risk of AD. , 1999), but the latter has not been conﬁrmed by others. This is an important area for future investigation.