Alzheimer's Disease: Advances in Etiology, Pathogenesis and by Khalid Iqbal (Editor), Sangram S. Sisodia (Editor), Bengt

By Khalid Iqbal (Editor), Sangram S. Sisodia (Editor), Bengt Winblad (Editor)

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Extra resources for Alzheimer's Disease: Advances in Etiology, Pathogenesis and Therapeutics

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This instrument assesses multiple cognitive abilities, including orientation as to time and place, immediate and delayed word recall, naming, verbal repetition, reading, writing, and spatial ability. , 2000). This result supports the view that the earliest cognitive deficits in AD are seen within the domain of episodic memory. 2. , 2001) reported the results from a comprehensive neuropsychological battery administered to a sample of the entire Kungsholmen Project’s cohort. This sample was examined three times during a follow-up period of six years.

Unfortunately this does not appear to be true. , 1997)] indicates that the exponential increase of the prevalence of dementia continues well past 85, apparently up to age 95. As the prevalence of dementia may exceed 50% after age 95, there must then be a plateau, since there certainly are centenarians with intact cognitive functions. Determining what happens at very advanced ages will be a major objective for future epidemiological studies. DIAGNOSING VERY MILD CASES OF DEMENTIA IN EPIDEMIOLOGICAL STUDIES Most of the prevalence studies cited have used either Mini-Mental State Examination (MMSE) scores of 24 or lower or equivalent scores on related tests as a screening tool.

VASCULAR RISK FACTORS Many clinicians have been struck by the seeming good health of individuals who develop AD. , 1990). Sparks et al. (1990) reported a high correlation between coronary stenosis and cerebral plaques in a medical examiner autopsy series. ApoE-e4 predisposes to myocardial infarcts in young adult males and AD later in life. , 1997) may alter both brain amyloid and the risk of AD. , 1999), but the latter has not been confirmed by others. This is an important area for future investigation.

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